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in college or gainfully returned to work. The premise of only taking the ICP in to account without weighing other factors is indicative of a complete patient mismanagement and delenquncy of the medical team doing so. Cou can not monitor ICP alone with out taking in to account the perfusion pressures(MAP-ICP=CPP) and end capillary brain tissue oxygenation(pbtO2)in the penumbra of the injury. Since 2002 any reputable medical center that has managed patients with elevated ICPs has done so knowing that they must maintain adequate cerebral perfusion. When the brain loses compliance the perfusion thresholds increase not entirely unlike someone with noncompliant lungs needing positive pressure ventilation to recruit alveoli and exchange gases. Guided brain tissue oxygenation and cerebral microdialysis are vanguard in assuring that these types of patients get the very best care. It's the brain, stupid!
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The whole premise that if your ICP goes beyond 40mm/hg you will suffer irrepairble damage is false. I have managed many TBI patients who have had sustained ICP's in the high 30's and 40's for days with decent recovery of functionl. Some, but not all, go on to work and function in society. Several are
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which might be appropriate (or interesting) to address in this page. The question is about the nonpathologic perception of increased pressure in the head from reclining with the head below the heart. I think that, for the average reader, an explanation of how pressure is normally regulated, and
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I'd be opposed to merging the two articles and leaving a redirect, because intracranial pressure and hydrocephalus are two completely different things. A lot of the head trauma pages link to ICP because of cerebral edema that results, e.g.
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is a stub. High ICP is discussed in much greater detail here. Alternately, we could merge the content on high ICP from here to there and keep two separate pages. I'd think a merge would be better though.
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Thanks for the fixes you've made so far. If you have some expertise in the field, please continue the good work, ideally supported by sources that meet the
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Unal-Cevik, I.; Arslan, D. (April 2023). "Similarities and differences between migraine and other types of headaches: Migraine mimics".
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Popa, Stefan L.; Chiarioni, Giuseppe; David, Liliana; Golea, George I.; Dumitrascu, Dan L. (25 March 2020). "Rare causes of emesis".
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When you have finished reviewing my changes, you may follow the instructions on the template below to fix any issues with the URLs.
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Good question. Boldly fixed that. I can carry out the merge, it won't take much because most of the info's already here.
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to delete these "External links modified" talk page sections if they want to de-clutter talk pages, but see the
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If you have discovered URLs which were erroneously considered dead by the bot, you can report them with
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on 10 July 2024. For the contribution history and old versions of the redirected page, please see
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adds anything more than isn't covered better here already. An alternative target might be
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This page needs work! Will try in coming months to sit down and write a full paper.~
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Bos, RF; Ramaker, C; van
Ouwerkerk, WJ; Linssen, WH; Wolf, BH (27 July 2002). "".
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in this article, which can be found in another article in the wikipedia here:
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by not to the extent of doing the hard work myself. ;-) Also, why does
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Just thought they should be linked. I'd do it if I knew how.
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As a symptom for eICP? I haven't found any citation for it.
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